论文代写：乳腺癌风险
PALB2基因被称为BRCA2的定位器和合作伙伴。该基因提供了产生蛋白质的方向，这些蛋白质与BRCA2结合，修复受损的DNA，阻止肿瘤的生长。异常的PALB2基因会增加患乳腺癌的风险。它增加了6到9倍的风险，几乎和异常的BRCA2或BRCA1基因一样高。PTEN基因有助于调节细胞的生长。异常的PTEN基因导致了一种考登综合症，在这种综合征中，人们有乳腺癌的风险，也有甲状腺、消化道、卵巢和子宫肿瘤的生长。RAD50基因与NBN和MRE11A基因形成MRN复合物，有助于修复细胞中的DNA损伤。
论文代写：乳腺癌风险
异常的RAD50基因与乳腺癌的风险有关，因为异常基因阻止细胞修复受损的DNA (Michell, 2010)。RDA51C基因修复受损的DNA。遗传到个体身上的异常基因具有较高的乳腺癌风险。STK11基因调控细胞的生长。异常的STK11基因导致Peutz Jegher综合征，这是一种罕见的疾病。它导致了一种被称为错构瘤的息肉的发展。这种息肉是在胃、结肠和小肠中发现的。有Peutz Jegher综合征的人有乳腺癌、肺癌、卵巢癌和胃肠道癌的风险。
论文代写：乳腺癌风险
The PALB2 genes are called the localizer and partner of BRCA2. The gene provides direction to produce proteins that work with BRCA2 to repair the damaged DNA and prevent the growth of the tumour. The abnormal PALB2 genes increase the risk of breast cancer. It increases the risk from 6 to 9 times which almost as high as the abnormal BRCA2 or BRCA1 genes. The PTEN genes help to regulate the growth of the cells. The abnormal PTEN genes cause a Cowden Syndrome in which people are at the risk of breast cancer and growths of the tumours in the thyroid, digestive tract, ovaries and uterus. The RAD50 genes along with the NBN and MRE11A genes form MRN complex that helps to repair the DNA damage in the cells.
论文代写：乳腺癌风险
The abnormal RAD50 genes are linked to the risk of the breast cancer because the abnormal genes prevent the cells to repair the damaged DNA (Michell, 2010). The RDA51C genes repair the DNA that is damaged. The abnormal genes that are inherited to the body of an individual are at the higher risk of breast cancer. The STK11 genes regulate the growth of the cells. The abnormal STK11 gene causes the Peutz Jegher syndrome which is a rare disorder. It leads to the development of a type of polyp which is known as a hamartomatous polyp. This polyp is found in the stomach, colon and small intestine. People who are having the Peutz Jegher Syndrome are at the risk of breast, lung, ovarian and gastrointestinal cancers.
